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In reply to the discussion: The legacy of Andrew Wakefield continues [View all]proverbialwisdom
(4,959 posts)57. INFO, Hekate and Sid. Take it or leave it, your choice.
RECOMMENDED: http://www.democraticunderground.com/10022587413
RECOMMENDED: http://www.democraticunderground.com/1017110012
Again, conclude what you wish. The future looks promising for sorting all this out, I'd say.
http://adventuresinautism.blogspot.com/2013/03/testimony-on-maine-ld-672-act-relating.html
By Ginger Taylor
March 11, 2013
...Vaccine policy should not be based merely on the reduction of communicable disease levels, but on overall health outcomes for children, including the true increased risk of autoimmune and neurological disorders that may be caused by an overaggressive and inappropriate vaccine schedule.
By Ginger Taylor
March 11, 2013
...Vaccine policy should not be based merely on the reduction of communicable disease levels, but on overall health outcomes for children, including the true increased risk of autoimmune and neurological disorders that may be caused by an overaggressive and inappropriate vaccine schedule.
RECOMMENDED: http://www.democraticunderground.com/1017110012
Here's a press release about a study recently published in a peer-reviewed journal. I added the underline in critical note #3. (FYI, Professor/Dr. John Walker-Smith is regarded as the co-founder of the field of pediatric gastroenterology with Harvard Professor/Dr. Allan Walker).
PLOS ONE Journal Information
PLOS ONE (eISSN-1932-6203) is an international, peer-reviewed, open-access, online publication. PLOS ONE welcomes reports on primary research from any scientific discipline. It provides:
Open-accessfreely accessible online, authors retain copyright
Fast publication times
Peer review by expert, practicing researchers
Post-publication tools to indicate quality and impact
Community-based dialogue on articles
Worldwide media coverage
http://www.jabs.org.uk/
CryShame Press Release - 9 March 2013
http://www.cryshame.co.uk
Important new research ( http://www.plosone.org/article/info:doi/10.1371/journal.pone.0058058 ) reports similar findings to the work of Dr Andrew Wakefield in the 1998 Lancet and in subsequent paper in the early 2000s
Groundbreaking new research examines the molecular structure of inflammatory material taken from the bowels of autistic children. It compares the structure of diseased biopsies in the autistic children with biopsies from three groups of non-autistic children with Crohns disease, ulcerative colitis, and histologically normal (the controls).
Previous research confirmed the pathological and immunological make-up of biopsies of autistic children, but had not to date identified its specific molecular structure. Children with the four different conditions have been found to have similar findings of inflammation. But it was not clear if this was the same condition shared by all four groups; or if a distinct condition was specific to autistic children alone; or if indeed there was no disease in the autistic group. A molecular analysis of the genetic structure found in the inflamed bowel tissue of children in each group would provide initial answers to these questions.
To date government and medical scientists continue to deny an association between autism and bowel disease. In the UK there is currently no research into the association between autism and chronic bowel disease. This has been the predicament since the government and medical profession waged a campaign to discredit research from the Royal Free Hospital led by Dr Andrew Wakefield in 1998 and the early 2000s that first identified the presence of bowel disease in autistic children.
Following years of denial from government and the medical profession, new research published in the leading online journal PLOS ONE confirms the presence of intestinal disease in autistic children and supports reports from many parents of ongoing painful gastric problems in their autistic children.
The research studied bowel samples from 25 autistic, 8 Crohn's, 5 ulcerative colitis and 15 normal control children and found that inflammatory material obtained from the biopsies of autistic children had a distinct molecular structure that was different from the other three groups.
This is an important finding of the distinct genetic expression that has now been identified in autistic children as distinct from non-autistic children with Crohns, ulcerative colitis and normal bowels. It paves the way for future research into the specific molecular structure of the inflammation affecting autistic children and hopefully will lead to new interventions and treatment.
Background Notes
1. The first paper to bring to public attention the presence of bowel disease in autistic children was Wakefield AJ, (1998) 'Ileal-lymphoid-nodular hyperplasia, non-specific colitis, and pervasive developmental disorder in children'.The Lancet published this paper in 1998 but subsequently retracted it in 2010 after the GMC found Dr Wakefield and Professor Walker-Smith guilty of serious professional misconduct.
2. Several former colleagues went on in the early 2000s to study the nature of the bowel disease in autistic children, focusing on the pathology of gut tissue and the presence of autoimmune features in the bowel (eg Furlano et al (2001) 'Colonic CD8 and ?? T-cell infiltration with epithelial damage in children with autism', Journal of Pediatrics, Vol. 138, 3).
3. The senior research leader of the Lancet and subsequent papers was Professor John Walker-Smith who in March 2012 had all the charges of professional misconduct made by the GMC quashed on appeal by Justice Mitting in the High Court.
4. Government Minister admits more needs to be done to research autism and bowel disease. Read letter here.
CryShame Press Release - 9 March 2013
http://www.cryshame.co.uk
Important new research ( http://www.plosone.org/article/info:doi/10.1371/journal.pone.0058058 ) reports similar findings to the work of Dr Andrew Wakefield in the 1998 Lancet and in subsequent paper in the early 2000s
Groundbreaking new research examines the molecular structure of inflammatory material taken from the bowels of autistic children. It compares the structure of diseased biopsies in the autistic children with biopsies from three groups of non-autistic children with Crohns disease, ulcerative colitis, and histologically normal (the controls).
Previous research confirmed the pathological and immunological make-up of biopsies of autistic children, but had not to date identified its specific molecular structure. Children with the four different conditions have been found to have similar findings of inflammation. But it was not clear if this was the same condition shared by all four groups; or if a distinct condition was specific to autistic children alone; or if indeed there was no disease in the autistic group. A molecular analysis of the genetic structure found in the inflamed bowel tissue of children in each group would provide initial answers to these questions.
To date government and medical scientists continue to deny an association between autism and bowel disease. In the UK there is currently no research into the association between autism and chronic bowel disease. This has been the predicament since the government and medical profession waged a campaign to discredit research from the Royal Free Hospital led by Dr Andrew Wakefield in 1998 and the early 2000s that first identified the presence of bowel disease in autistic children.
Following years of denial from government and the medical profession, new research published in the leading online journal PLOS ONE confirms the presence of intestinal disease in autistic children and supports reports from many parents of ongoing painful gastric problems in their autistic children.
The research studied bowel samples from 25 autistic, 8 Crohn's, 5 ulcerative colitis and 15 normal control children and found that inflammatory material obtained from the biopsies of autistic children had a distinct molecular structure that was different from the other three groups.
This is an important finding of the distinct genetic expression that has now been identified in autistic children as distinct from non-autistic children with Crohns, ulcerative colitis and normal bowels. It paves the way for future research into the specific molecular structure of the inflammation affecting autistic children and hopefully will lead to new interventions and treatment.
Background Notes
1. The first paper to bring to public attention the presence of bowel disease in autistic children was Wakefield AJ, (1998) 'Ileal-lymphoid-nodular hyperplasia, non-specific colitis, and pervasive developmental disorder in children'.The Lancet published this paper in 1998 but subsequently retracted it in 2010 after the GMC found Dr Wakefield and Professor Walker-Smith guilty of serious professional misconduct.
2. Several former colleagues went on in the early 2000s to study the nature of the bowel disease in autistic children, focusing on the pathology of gut tissue and the presence of autoimmune features in the bowel (eg Furlano et al (2001) 'Colonic CD8 and ?? T-cell infiltration with epithelial damage in children with autism', Journal of Pediatrics, Vol. 138, 3).
3. The senior research leader of the Lancet and subsequent papers was Professor John Walker-Smith who in March 2012 had all the charges of professional misconduct made by the GMC quashed on appeal by Justice Mitting in the High Court.
4. Government Minister admits more needs to be done to research autism and bowel disease. Read letter here.
PLOS ONE Journal Information
PLOS ONE (eISSN-1932-6203) is an international, peer-reviewed, open-access, online publication. PLOS ONE welcomes reports on primary research from any scientific discipline. It provides:
Open-accessfreely accessible online, authors retain copyright
Fast publication times
Peer review by expert, practicing researchers
Post-publication tools to indicate quality and impact
Community-based dialogue on articles
Worldwide media coverage
Again, conclude what you wish. The future looks promising for sorting all this out, I'd say.
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Amen. My local austism society chapter invited this jerk to speak AFTER his license
Butterbean
May 2013
#17
"Trifles make perfection (or science), but perfection is no trifle." - Michelangelo
proverbialwisdom
May 2013
#16
Ignore them, read their links to UK NHS data and reach your own conclusions.
proverbialwisdom
May 2013
#21
When your kid is diagnosed with autism, you grasp onto any simple explanation
Canuckistanian
May 2013
#24
Uh, no, straw man fallacies beginning with the unflawed studies paragraph. nt
proverbialwisdom
May 2013
#25
Smear away. You still haven't shown even once where I was wrong about the BFEE. Not even once.
Octafish
May 2013
#31
No. I didn't write that. Yet, you insist on associating me with something I did not write.
Octafish
May 2013
#34
I've read the stat that 1 in 8 children of Somali immigrants in Minnesota are diagnosed with autism.
proverbialwisdom
May 2013
#38
KARE 11 TV Minneapolis: “1 in 8 kids in the local Somali community are affected” (VIDEO)
proverbialwisdom
May 2013
#53
This speaks for itself in correcting a few of the misrepresentations on this thread.
proverbialwisdom
May 2013
#39
AOA is an INTERMEDIARY between primary peer-reviewed material and the public vetted by SMART parents
proverbialwisdom
May 2013
#43
New study by Dr. Martha Herbert & Dr. Julie Buckley in Journal of Child Neurology on autism and diet
proverbialwisdom
May 2013
#44
Absolutely misleading, if true factoid, and the Journal of Child Neurology is peer-reviewed.
proverbialwisdom
May 2013
#48
"Nothing of value in terms of original work or trying to interpret results from other places," oh?
proverbialwisdom
May 2013
#49
Wakefield lost his medical license for using kids as subjects with "callous disregard"
Hekate
May 2013
#52
In the '60s I knew a girl who'd had mumps encephalitis. She was blind and crippled.
Hekate
May 2013
#54
Check it out, please. Video features GR Executive Director Candace McDonald and her brother.
proverbialwisdom
May 2013
#62