You hear antivaxers use "VAERS" a lot

[ARPad95]

2. "Moderna gave her herpes"

Last edited Tue Jan 4, 2022, 03:29 PM - Edit history (2)

I'd guess she had been infected with herpes sometime in her past and vaccination caused it to reactivate. In other words, vaccination did NOT give her herpes.

https://onlinelibrary.wiley.com/doi/10.1111/jdv.17646

Although the precise mechanism is not known, herpesvirus reactivation may occur due to innate- or cell-mediated immune defense failures initiated by the host response to vaccination.

Cytomegalovirus (CMV) is in the herpes family of viruses* and most adults were infected with it previously (as early as young childhood). It lies dormant in the body and may reactivate whenever the immune system is suppressed or compromised.

https://www.ncbi.nlm.nih.gov/books/NBK8157/

General Biology of Human Herpesviruses

Of the more than 100 known herpesviruses, 8 routinely infect only humans: herpes simplex virus types 1 and 2, varicella-zoster virus, cytomegalovirus, Epstein-Barr virus, human herpesvirus 6 (variants A and B), human herpesvirus 7, and Kaposi's sarcoma virus or human herpesvirus 8. A simian virus, called B virus, occasionally infects humans. All herpesviruses can establish latent infection within specific tissues, which are characteristic for each virus.

https://pubmed.ncbi.nlm.nih.gov/23611298/

Herpesviruses and the microbiome

Abstract

The focus of this article will be to examine the role of common herpesviruses as a component of the microbiome of atopic patients and to review clinical observations suggesting that atopic patients might be predisposed to more severe and atypical herpes-related illness because their immune response is biased toward a TH2 cytokine profile. Human populations are infected with 8 herpesviruses, including herpes simplex virus HSV1 and HSV2 (also termed HHV1 and HHV2), varicella zoster virus (VZV or HHV3), EBV (HHV4), cytomegalovirus (HHV5), HHV6, HHV7, and Kaposi sarcoma-associated herpesvirus (termed KSV or HHV8). Herpesviruses are highly adapted to lifelong infection of their human hosts and thus can be considered a component of the human "microbiome" in addition to their role in illness triggered by primary infection.