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Bennyboy

(10,440 posts)
12. Thanks for that...... (From that article some conclusions.....)
Tue Dec 4, 2012, 03:14 PM
Dec 2012

The foregoing comparisons might be misleading. THC's effects differ qualitatively from many other drugs, especially alcohol. For example, subjects drive faster after drinking alcohol and slower after smoking marijuana (Hansteen et al., 1976/ Casswell, 1979; Peck et al., 1986; Smiley et al., 1987).. Moreover, the simulator study by Ellingstad et al. (1973) showed that subjects under the influence of marijuana were less likely to engage in overtaking maneuvers, whereas those under the influence of alcohol showed the opposite tendency. Very importantly, our city driving study showed that drivers who drank alcohol over-estimated their performance quality whereas those who smoked marijuana under-estimated it. Perhaps as a consequence, the former invested no special effort for accomplishing the task whereas the latter did, and successfully. This evidence strongly suggests that alcohol encourages risky driving whereas THC encourages greater caution, at least in experiments. Another way THC seems to differ qualitatively from many other drugs is that the former users seem better able to compensate for its adverse effects while driving under the influence. Weil et al. (1968) were among the earliest authors who mentioned the possibility that marijuana users can actively suppress the drug's adverse effects. They presumed that THC's effects were confined to higher cortical functions without any general stimulatory or depressive effect on lower brain centers. According to them, the relative absence of neurological, as opposed to psychiatric, symptoms in marijuana intoxication suggests this possibility. More recently, Moskowitz (1985) concluded that the variety of impairments found after marijuana smoking could not be explained by decrements in sensory or motor functions which led him to hypothesize that some important central cognitive process is impaired by THC, without saying what it is. Identification of THC's site of action would greatly enhance our understanding of the drug's psychopharmacological effects.

Epidemiological research has shown that THC is infrequently detected in the blood of fatally injured drivers as the only drug present. In most cases alcohol is also detected. The effects of the combination of THC and alcohol on actual driving performance have never been studied in the presence of other traffic. Closed-course studies have shown that the effects of both drugs, when taken in combination, are generally additive (Atwood et al., 1981; Peck et al., 1986). This may only be so for those behaviors that are similarly affected by both rugs given separately. Closer examination of the combined use is warranted in those driving situations where both drugs produce qualitatively different effects. It may well be so that alcohol reduces drivers' insight or motivation to the point where they would no longer attempt to compensate for the THC effect. As a result, the combined effects on drivers' performance could well be greater than the sum of either drug acting separately. There is therefore a great need for further research on marijuana and actual driving research, but now extended to the combination of marijuana and alcohol.

In summary, this program of research has shown that marijuana, when taken alone, produces a moderate degree of driving impairment which is related to the consumed THC dose. The impairment manifests itself mainly in the ability to maintain a steady lateral position on the road, but its magnitude is not exceptional in comparison with changes produced by many medicinal drugs and alcohol. Drivers under the influence of marijuana retain insight in their performance and will compensate where they can, for example, by slowing down or increasing effort. As a consequence, THC's adverse effects on driving performance appear relatively small

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