The problem with glioblastoma is that completely removing all the visible tumor --as the surgeon did

[KewlKat]

33. If you'd care to read about it here are a couple. Second article talks about why US doesnt trial

Metabolic management of glioblastoma multiforme using standard therapy together with a restricted ketogenic diet: Case Report

Results

After two months treatment, the patient's body weight was reduced by about 20% and no discernable brain tumor tissue was detected using either FDG-PET or MRI imaging. Biomarker changes showed reduced levels of blood glucose and elevated levels of urinary ketones. MRI evidence of tumor recurrence was found 10 weeks after suspension of strict diet therapy.

Conclusion

This is the first report of confirmed GBM treated with standard therapy together with a restricted ketogenic diet. As rapid regression of GBM is rare in older patients following incomplete surgical resection and standard therapy alone, the response observed in this case could result in part from the action of the calorie restricted ketogenic diet. Further studies are needed to evaluate the efficacy of restricted ketogenic diets, administered alone or together with standard treatment, as a therapy for GBM and possibly other malignant brain tumors.


https://nutritionandmetabolism.biomedcentral.com/articles/10.1186/1743-7075-7-33


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The Restricted Ketogenic Diet: An Alternative Treatment Strategy for Glioblastoma Multiforme

Conclusions

We provide information on a new, alternative treatment strategy for GBM
management that targets tumor energy metabolism. The objective of
this new therapeutic strategy is to change the metabolic environment of
the tumor and the host. Access to glucose and glutamine within the
tumor microenvironment provides neoplastic GBM cells and associated
host stromal cells (macrophages) with fermentable fuels necessary for
maintaining the growth and survival of malignant brain tumor cells in the
hypoxic microenvironment. The low-carbohydrate, high-fat ketogenic
diet, will reduce circulating glucose levels and will elevate circulating
levels of ketone bodies especially when consumed in restricted amounts
(RKD). A transition from glucose to ketone bodies will restrict glucose
availability to the malignant tumor cells while protecting and enhancing
the health and vitality of normal brain cells. The therapeutic efficacy of
the RKD against GBM can be enhanced when combined with drugs that
also target or reduce access to glucose and glutamine. A use protocol is
presented for assisting physicians and GBM patients in implementing the
RKD as a treatment strategy. Although the RKD is less toxic and
potentially more effective in managing GBM than the conventional
standard of care, considerable patient education, motivation, and
discipline will be necessary for implementing this therapy. Considering
the unfavorable outcome of most GBM patients treated with the current
standard of care, the RKD treatment strategy could be an attractive
treatment option for some GBM patients.

http://www.rsg1foundation.com/docs/patient-resources/The%20Restricted%20Ketogenic%20Diet%20An%20Alternative.pdf