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TexasProgresive

(12,740 posts)
4. I have no disagrement with your post.
Mon Mar 3, 2014, 07:47 AM
Mar 2014

My point is that type 2 is not caused by the destruction of the islets of Langerhans (beta)insulin producing cells as suggested in the article by any means whether autoimmune response or some kind of inflammation. Below are 2 excerpts from the NIH on both type 1 and 2 diabetes. Neither makes mention of "to be caused by oxidation in the body that causes inflammation and kills off pancreatic cells." I think Dr. Watson is on the wrong foot in this matter.

What causes type 1 diabetes?

Type 1 diabetes is caused by a lack of insulin due to the destruction of insulin-producing beta cells in the pancreas. In type 1 diabetes—an autoimmune disease—the body’s immune system attacks and destroys the beta cells. Normally, the immune system protects the body from infection by identifying and destroying bacteria, viruses, and other potentially harmful foreign substances. But in autoimmune diseases, the immune system attacks the body’s own cells. In type 1 diabetes, beta cell destruction may take place over several years, but symptoms of the disease usually develop over a short period of time.


What causes type 2 diabetes?

Type 2 diabetes—the most common form of diabetes—is caused by a combination of factors, including insulin resistance, a condition in which the body’s muscle, fat, and liver cells do not use insulin effectively. Type 2 diabetes develops when the body can no longer produce enough insulin to compensate for the impaired ability to use insulin. Symptoms of type 2 diabetes may develop gradually and can be subtle; some people with type 2 diabetes remain undiagnosed for years.

Type 2 diabetes develops most often in middle-aged and older people who are also overweight or obese. The disease, once rare in youth, is becoming more common in overweight and obese children and adolescents. Scientists think genetic susceptibility and environmental factors are the most likely triggers of type 2 diabetes.

http://diabetes.niddk.nih.gov/dm/pubs/causes/

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