A better assessment of the OP piece.
http://theness.com/neurologicablog/index.php/non-celiac-gluten-sensitivity/
"A recent study suggests that this might be the case. Biesiekierski et. al. did a well controlled series of studies in which they challenged subjects with possible NCGS with carefully controlled diets with various amounts of gluten. They found no association between gluten consumption and reported symptoms, arguing very strongly against NCGS as a real entity.
Their study did, however, suggest another possible culprit – FODMAPs (fermentable, oligo-, di-, monosaccharides, and polyols). These are also common in breads and other foods containing gluten. In the study subjects, GI symptoms improved when FODMAPs and gluten were removed, but then reintroducing gluten had no association with return of symptoms. The authors conclude:
"In a placebo-controlled, cross-over rechallenge study, we found no evidence of specific or dose-dependent effects of gluten in patients with NCGS placed diets low in FODMAPs."
They were not, however, testing whether or not FODMAPs were a cause of GI symptoms, and so cannot conclude if this is the true cause. A follow up study would need to be done to verify that (perhaps we’ll see a FODMAP-free fad before this science can be done). If true it would explain why some people do have reduction in GI symptoms when they avoid gluten, because they are also avoiding FODMAPs.
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